If we have another Biden presidency our nation is done. The globalists at the WEF and the enemies of America are using this corrupt POS to infiltrate and destroy our nation. Donald Trump is our best bet moving forward but there are a LOT of people very interested in infiltrating his inner circle and undermining what he's trying to do. To that extent we have some serious questions about Trump's main gatekeeper and the head of his PAC. This person is ideally positioned to cause trouble and is working directly with Pfizer, Gilead, and GAVI. MAGA needs to support the real president by helping watch for infiltrators. 

Rumble: 

My Twitter/X posts on Susie Wiles:

BREAKING: Top Trump Advisor is Working for Consulting Company That Also Supports Democrats and is Owned by a Company Tied to WEF: I absolutely support Donald Trump for President but there is a snake in the woodshed. Unless @realDonaldTrump is surrounded by advisors that are selling out I cannot explain his support for Johnson and other RINOs. @SpeakerJohnson is a sellout and Trump’s continued support for #RINOs that will absolutely throw him under the bus is beyond my comprehension. Of course when his top advisors are partners in companies that represent Pfizer, Gilead & GAVI in a company owned by the #WEF I can see why he’d be getting bad advice. Maybe people like Suzy should not be advising the President on what the #Trump2024 crowd wants… just saying but mRNA fans usually are sellouts. We the People MUST help Trump watch for sellouts… maybe I’m wrong but this deserves investigation. More in the comments but I’m tired of the support for people stabbing America in the back.

Link: https://x.com/RenzTom/status/1782225573193855215

 

 

 

 

Susie Wiles (the #Trump2024 campaign manager, pac leader, gatekeeper to @realDonaldTrump & partner in Mercury LLC - a company representing Democrats Pfizer Gilead GAVI the UN Foundation & tied to the WEF) is apparently NOT the only person we need to be investigating around Trump. If #WeThePeople of MAGA want to win we need to help Trump drain the swamp around him. This is critical and systemic. Thank you Alex Jones for helping me get this out & keep an eye on @drdrew this evening for more.

Link: https://x.com/RenzTom/status/1783120183755747432

https://x.com/MailOnline/status/1783097752030740968

https://nationalfile.com/susie-wiles-who-oversees-trumps-endorsements-is-co-chair-of-firm-managed-by-recent-pfizer-vice-president/

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Thank you for joining me again as I continue to examine, without bias - the different theories on the origins, development and treatment of cancer. If you missed my previous Substacks in this cancer series, please feel free to check those out here and here.

An increasingly popular, although admittedly not mainstream theory - is that cancer is a metabolic disease. This article is not intended to be a comprehensive presentation of the theory but intended to serve as a basic overview to introduce you to the subject. Much like the theory that some cancers are parasitic in nature, the idea that cancer is a metabolic disease - is not widely examined or acknowledged by the mainstream medical system. Outspoken advocates in support of the theory assert that this approach is not being recognized as an effective alternative to the radiation and chemotherapies that are currently being utilized - because there is little money to be made.

Chemotherapy and radiation bring in billions of dollars in revenue and some experts believe that the financial incentives to continue to recommend these treatments keep the scale tipped in their favor despite the fact that they are extremely toxic. One is a money generating approach, the other is not - so why would they engage in research that could potentially cut into (or decimate) the profits of a multibillion-dollar industry? Further, while chemo and radiation can be very effective, the side effects can cause a number of other issues including an increased likelihood of recurrence of cancer. Cancer does kill and chemo/radiation do frequently work well to stop it but what if there is another way that had fewer side effects? Drugs or supplements that could cure cancer without the need for other drugs to combat their side effects? It is certainly unseemly to discuss but it is inarguable that it would be a financial disaster for those making money from cancer if something like that were to become available.

Many physicians and oncologists are completely unfamiliar with the concept of cancer as a complex metabolic disease. It’s simply not taught, or it’s just dismissed outright. Most cancer research today is focused on gene mutations and the concept of “personalized medicine” in cancer involves the development of drugs and things like mRNA gene therapies. These gene therapies and drugs are being developed - presumably -to target specific mutations that are found in various tumors - with the hope of correcting the disease.

Some experts, like Dr. Thomas Seyfried will tell you that the problem with that is that there are thousands, possibly millions of mutations in tumor cells which makes targeting them a daunting task that comes with toxic adverse effects. Dr. Seyfried received his Ph.D. in Genetics and Biochemistry from the University of Illinois and boasts impressive credentials. He published Cancer as a Metabolic Disease: On the Origin, Management, and Prevention of Cancer and in his book, Dr. Seyfried presents an alternative origin of cancer based on the theories of Dr. Otto Warburg, who found that the root cause of cancer is a combination of low cellular oxygenation (hypoxia) and acidosis. In his “On the Origins of Cancer Cells” publication Warburg summarized that “the cause of cancer is the replacement of the respiration of oxygen in normal body cells by a fermentation of sugar.” Normal body cells meet their energy needs by respiration of oxygen, whereas cancer cells meet their energy needs - in great part - by fermentation.

Warburg showed that cancer cells are fermenters of glucose. Dr. Seyfried expands on that theory and asserts that cancer is a disease of cellular metabolic dysfunction due to damaged mitochondria.

 

 

He claims that glucose is metabolized to the point of pyruvate which is then fermented. Almost all cancer cells have this property because tumor cells must ferment either glucose (or amnio acids like glutamine) for survival and growth since they are anaerobic and cannot survive without oxygen.

Dr. Seyfried concludes that cancer is not a genetic disease and that the mutations are merely ‘downstream epiphenomenon of damaged respiration’. Simply put, he asserts that the genetic mutations are red herrings caused by mitochondria that have suffered some sort of insufficiency or damage. If the mitochondria are healthy - you will not get cancer. Different chemicals, radiation, toxins, inflammation, hypoxia, viruses and inherited genes can damage functionality of mitochondria.

Cell Death & Disease published ‘Hypoxia Regulates the Mitochondrial Activity of Hepatocellular Carcinoma Cells through HIF/HEY1/PINK1 Pathway’ and revealed the following:

“Hypoxia is commonly found in cancers. Hypoxia, due to the lack of oxygen (O₂) as the electron recipient, causes inefficient electron transfer through the electron transport chain at the mitochondria leading to accumulation of reactive oxygen species (ROS) which could create irreversible cellular damages.”

A 2023 review ‘Hypoxic Microenvironment in Cancer: Molecular Mechanisms and Therapeutic Interventions tells us that ‘hypoxia damages the cellular genome and drives carcinogenesis’.

“Hypoxia plays a central role in cancer progression and resistance to therapy by promoting various changes in the biology of stromal cells in the TME, including immune cells.”

Ask yourself, how much does genetic manipulation (gene therapy) affect the functionality of mitochondria? What is the effect of inserting experimental mRNA (modRNA) into the genes of the recipients and their offspring?

The metabolic theory of cancer claims glucose, oxidative stress, and mitochondrial health affect the onset and spread of the disease. In addition to pointing to new directions of research, Dr. Seyfried elaborates on a non-toxic mode of treatment, therapeutic fasting and a ketogenic diet. The ketogenic diet is a low carb/high fat diet and was originally used as one of the first treatments of epilepsy in children. The ketogenic diet (KD) capitalizes on the inability of the damaged cancer cell mitochondria to metabolize ketones, thus starving them while maintaining healthy cells.

Because cancer cells develop quickly on glucose and glutamine, it has been demonstrated that a ketogenic diet can effectively deprive malignant cells of glucose. Ketogenic diets have a fairly straightforward concept. In the simplest way I can explain it - if glucose serves as the main fuel for cancer, then reducing your consumption of carbohydrates and substituting them with other fuels, like fats, will force your body to enter a state of ketosis. Tumors that are fueled by glucose will have difficulty trying to compete with normal cells for the restricted source of glucose. Tumor cells can’t utilize the ketones for energy like the normal, healthy cells can. The body should be restricted from the primary fuels for the tumor while the body transitions to the ketones to fuel the body. Simply put, if you starve the malignant cells of its fuel, the theory is that it will kill tumor cells.

 

A paper published by Seyfried et al in BMC ‘Press-pulse: A Novel Therapeutic Strategy for the Metabolic Management of Cancer’ concluded the following:

“Hyperbaric oxygen therapy combined with the calorie restricted ketogenic diet will kill tumor cells through apoptotic and anti-angiogenic mechanisms while also reducing inflammation in the tumor microenvironment and systemically.”

A 2021 NIH publication called ‘Ketogenic Diet in Cancer Prevention and Therapy: Molecular Targets and Therapeutic Opportunities’ documented the following:

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“The ketogenic diet (KD), a high-fat and very-low-carbohydrate with adequate amounts of protein, has shown antitumor effects by reducing energy supplies to cells. This low energy supply inhibits tumor growth, explaining the ketogenic diet’s therapeutic mechanisms in cancer treatment.”

A 2015 NIH study entitled ‘Non-Toxic Metabolic Management of Metastatic Cancer in VM Mice: Novel Combination of Ketogenic Diet, Ketone Supplementation, and Hyperbaric Oxygen Therapy’ published some interesting findings.

“Mice receiving the combination therapy demonstrated a marked reduction in tumor growth rate and metastatic spread and lived twice as long as control animals. To further understand the effects of these metabolic therapies, we characterized the effects of high glucose (control), low glucose (LG), ketone supplementation (βHB), hyperbaric oxygen (HBOT), or combination therapy (LG+βHB+HBOT) on VM-M3 cells. Individually and combined, these metabolic therapies significantly decreased VM-M3 cell proliferation and viability. HBOT, alone or in combination with LG and βHB, increased ROS production in VM-M3 cells. This study strongly supports further investigation into this metabolic therapy as a potential non-toxic treatment for late-stage metastatic cancers.”

Drugs like Gleevec and Herceptin target the same metabolic pathway that the ketogenic diet does. The difference is - a ketogenic diet theoretically targets all cancers whereas the drug specific ones - target those pathways only in the individuals with that mutation. Some experts are asking - why use toxic drugs when there is another alternative to do the same thing without toxicity to the body? Why are we attempting to genetically modify people before we attempt something as simple as a ketogenic diet? Please note - these are the experts theories - I’ll get to my thoughts in a moment.

A 2021 NIH paper ‘Ketogenic Diet for Cancer: Critical Assessment and Research Recommendations’ summarizes the strength of evidence of beneficial effects of KD for cancer related outcomes in pre-clinical and clinical studies.

 

The paper lists the following research recommendations for clinical studies examining the effects of KD on cancer development and progression:

 

• Conduct small, rigorous non-randomized trials with homogeneous patient groups and common cancer sites to assess whether KD produces a “signal” on selected outcomes (particularly those related to response to standard care (e.g., effectiveness, side effects)) that would justify the conduct of larger, randomized-controlled trials.

• In randomized-controlled trials, provide sufficient detail of the KD and control diets (ensuring that they are comparable on vitamins, mineral and other nutrients) so they could be replicated by other investigators.

• Develop a standardized method to monitor and quantify adherence and tolerance to the KD.

• Develop a set of standardized assessments and outcome measures that include the full array of relevant variables (e.g., imaging of tumor characteristics, body composition, quality of life, and survival).

• Distinguish trials based on whether they attempt to isolate the unique effects of KD versus those which seek to estimate its effects as an adjunctive therapy.

• Examine the effects of exogenous ketones, alone and in conjunction with a KD, to determine whether they have synergistic or additive effects.

• Because it is unlikely that KD will cure cancer, trials should focus on whether KD reduces cancer progression or recurrence in those who experience remission through standard care.

It’s worth asking - is progress is not being made in regard to curing cancer because they’re focusing on the wrong things?

Ultimately I believe the metabolic theory, much like the parasitic theory of cancer makes sense but probably does not explain every cancer, nor would it fix every cancer. The reason I say this is that I do not believe we are still dealing with cancer as a natural phenomenon. Cancer as a genetic issue certainly appears credible in many cases. I think many of these cancers stemming from genetic mutations will eventually be understood to have come from the poisons we are putting in our systems today; poisons that did not exist centuries ago.

The mRNA poisons are a great example. These gene therapy poisons and many other things we are putting in our body have a substantial impact on the genetic material and the functionality of our cells. Radiation can cause cellular mutation and every single radio signal and cellular tower in this country emit low levels of radiation and we have not properly studied their cancer causing potential. Modern life is full of things that may account for a major increase in cancer that is truly based on genetic mutation rather than a parasite or metabolic issue… or maybe these things contribute to metabolic issues in some way.

After researching and putting out these articles on cancer and causes I frankly am at the point I believe all three major theories of cancer seem credible. I am also appalled to see that only one of them is studied and that is the theory that makes the money. If we want to understand cancer we need to start by getting money out of the picture. Cancer kills and should not be political and should not be about money - but it is. I think it may be time we do something about that… more to come.

If you’d like to hear more on cancer as a metabolic disease the FLCCC Alliance published a webinar with Dr. Paul Marik and Dr. Thomas Seyfried that you can watch here.

Sources:

1. Clinical Trial Evidence Supporting US Food and Drug Administration Approval of Novel Cancer Therapies Between 2000 and 2016 - PubMed (nih.gov)

2. Cancer as a Metabolic Disease: On the Origin, Management, and Prevention of Cancer: 9780470584927: Medicine & Health Science Books 

   [email protected]

3. Cancer: An Unbiased Overview in Preparation for Our Upcoming War on Cancer (substack.com)

4. Mitochondria - Definition, Function & Structure | Biology Dictionary

5. Home - Thomas Seyfried (tomseyfried.com)

6. Cancer as A Metabolic Disease - FLCCC Alliance (covid19criticalcare.com)

7. Hypoxia regulates the mitochondrial activity of hepatocellular carcinoma cells through HIF/HEY1/PINK1 pathway | Cell Death & Disease (nature.com)

8. HIF-1α-induced expression of m6A reader YTHDF1 drives hypoxia-induced autophagy and malignancy of hepatocellular carcinoma by promoting ATG2A and ATG14 translation | Signal Transduction and Targeted Therapy (nature.com)

9. Press-pulse: a novel therapeutic strategy for the metabolic management of cancer | Nutrition & Metabolism | Full Text (biomedcentral.com)

10. Non-Toxic Metabolic Management of Metastatic Cancer in VM Mice: Novel Combination of Ketogenic Diet, Ketone Supplementation, and Hyperbaric Oxygen Therapy - PMC (nih.gov)

11. Ketogenic Diet in Cancer Prevention and Therapy: Molecular Targets and Therapeutic Opportunities - PMC (nih.gov)

12. chemotherapy-spreading-cancer-pdf.pdf (mercola.com)

13. Johns Hopkins Medicine Celebrates Its Contributions to Keto Therapy as Diet Turns 100 | Johns Hopkins Medicine

14. Gleevec: Uses, Dosage, Side Effects - Drugs.com

15. Ketogenic Diet for Cancer: Critical Assessment and Research Recommendations - PMC (nih.gov)

16. Herceptin (Trastuzumab): Side Effects, How it Works, and More (breastcancer.org)

     

     

    Subscribe: Join us on Substack for access to all of Tom's extremely detailed and mostly boring corruption fight research/data/sources/court docs... it's alot.. TomRenz.Substack.com

     

As a compendium to my previous substack on cancer, I offer you a brief overview of the theory that cancer is parasitic in origin. Ultimately I believe theory may apply in some cases but not all. Further, since everything big pharma and mainstream medicine summarily dismisses this theory I think it should be formally studied and proved or disproved so we know. This is not intended to be a comprehensive detailing, it’s simply an introduction. Affording thereto, it’s not possible to cover every expert so if I didn’t include something you believe is relevant - leave me a comment below.

In the mainstream, parasite infection is a commonly disregarded cancer risk factor even though research shows a feasible link between parasites and the development of cancer. Despite the relationship between parasites and other disease, including cancer this theory is often dismissed by ‘big medicine’. Some experts speculate that the reason for this rebuff is clear: money. Simply put, there is no money to be made in a medical system that exists in a parasite paradigm versus a ‘virus’ paradigm. Anti-parasitic medications like Ivermectin and Fenbendazole are pennies on the dollar compared to costly alternatives like chemotherapy and radiation. One treatment option buys the doctor a summer home - while the other does not. The financial incentives to keep such a revelation from being endorsed -or even acknowledged by the system is in the billions, if not more. Their entire industry could topple if people were to learn that the source of their disease could be so simple.

The Center for Disease Controls defines parasite as “an organism that lives on or in a host organism and gets its food from or at the expense of its host. They thrive off of foods such as dairy products, sugar and protein. They list three main classes of parasites that can cause disease in humans: protozoa, helminths, and ectoparasites.

 

Cancer is characterized by the unchecked growth of aberrant and altered cells that have the capacity to invade nearby tissues. Which, according to some experts is exactly what some parasites do within the body.

Dr. Lee Merritt has served on the Board of the Arizona Medical Association and is a past president of the Association of American Physicians and Surgeons (AAPS). She proposes, in the simplest way I can summarize it - that parasites in the body slowly increase in number over a lifetime and that they can lay thousands, upon thousands of eggs a day and form intracellular cysts. As more cysts present themselves it takes more of your immune system to keep them in check. The more active the immune system, the more likely you are to experience autoimmune disease. She postulates that when their numbers get too many, when these microscopic invaders are out of control and egg sacs enlarge and invade nearby tissues- this is when we see metastasis. Metastasis is when cancer has spread to a different part of the body part than where it started. Cancer spreads this way via the lymphatic system and it’s plausible to picture these invaders traveling throughout the body wreaking havoc.

In 1890 Scottish microbiologist and pathologist William Russell reported finding parasitic spores within cancer cells. He published his findings in the British Medical Journal and went on to formulate a comprehensive hypothesis explaining how cancer is a parasitic disease. As previously mentioned, these theories have routinely been dismissed within the medical community which remains steadfast in the ‘virus’ paradigm.

 

I recommend you read ‘How Parasites Get Their Hooks into Human Cells’, published by the Whitehead Institute for biomedical research. It explains the chain of cellular events that see parasites replicate and travel within cells and how other factors like calcium play a role.

“Whitehead Institute Member Sebastian Lourido studies apicomplexan biology using the species Toxoplasma gondii (T. gondii), which causes toxoplasmosis, a disease that can be deadly in people with compromised immune systems.

Active T. gondii parasites transition between two modes: replicating inside of one host cell and spreading to new cells. The transition between these modes happens very quickly: parasites will be busy replicating and then will rapidly exit the host cell and travel to new cells to invade them. Once inside a new cell, the parasites can switch back to reproducing or enter into a chronic stage—a hardy, inert form that can hide from the host’s immune system indefinitely.

Researchers knew that T. gondii starts its transition from the reproductive to the spreading mode with a calcium signal within the parasite. Calcium is an important signal that many cells use to trigger the quick release of different molecules, such as neurotransmitters from neurons and insulin from pancreatic cells in humans. In T. gondii, calcium is the signal that triggers release of molecules parasites need to exit host cells, travel, and invade. These include molecules to make the host cell membrane permeable and sticky molecules that the parasite uses to attach to and glide along host tissue, enabling it to move in the environment outside of the host cell. These molecules are packaged in small membrane-bound containers called micronemes, hundreds of which are scattered throughout the front third of the parasite cell.

The calcium signal begins a chain of signaling, like a game of telephone, that ultimately triggers the parasite cell to release the contents of these microneme packages. The cell does this by attaching the packages to a cellular conveyor belt system that runs along microtubules, part of the cytoskeleton. This whizzes them to the very front of the parasite, where their contents can be released outside of the parasite through a process called exocytosis, in which the membrane of the package fuses with the membrane of the parasite cell.”

A review published in Science Direct “Helminths in Human Carcinogenesis" reveals the following:

“Johanes Fibiger won the only Nobel Prize for helminthology in 1926 for the induction of gastric cancer in rats by feeding them cockroaches infected with Spiroptera neoplastica larvae (Campbell, 1997). The idea of parasitic worms causing cancer was not new. Fibiger had been aware of the causal relationship of Schistosoma haematobium and human urinary bladder cancer. His dissection of wild rats had yielded remarkable finding—stomach tumours containing worms.

Fibiger's worm, S. neoplastica (later renamed Gongylonema neoplastica), provided a unique scientific tool for the experimental induction of cancer in mammalian hosts. Fibiger's work was later criticized for the gastric tumours not being true cancers, but merely worm‐induced hyperplasia associated with vitamin A deficiency. Today, the International Agency for Research on Cancer (IARC) has in part vindicated Fibiger's belief in parasite‐induced cancer by labelling S. haematobium and Opisthorchis viverrini as definitely carcinogenic (group 1) and Clonorchis sinensis as probably carcinogenic (group 2) to humans (IARC, 1994).”

In November 2015, a study published in the New England Journal of Medicine called ‘Malignant Transformation of Hymenolepis nana in a Human Host’, revealed a shocking case in which cancer developed into a different kind of infectious parasite as a result of a tapeworm infection. Researchers found tapeworm DNA in the cancer cells when they looked at the immunocompromised patient's lymph node tissue.

 

If you’re thinking it’s only as deep as ‘people with compromised immune systems are just more susceptible to parasites’ - that’s fair. After all, correlation does not necessarily equal causation, however, it has been demonstrated that the tick-borne parasite T. parva creates a disease that is almost exactly like lymphoma, and that inoculating mice with the C. parvum parasite causes colon cancer abruptly.

Additionally, the “Helminths in Human Carcinogenesis" reveals the following:

“The International Agency for Research on Cancer (IARC) considered three species of trematodes, Schistosoma haematobium, Opisthorchis viverrini and Clonorchis sinensis, responsible for helminth induced human cancer; they considered S. haematobium and O. viverrini as group 1 carcinogens and C. sinensis as a group 2 carcinogen [2].

Helminth infections are of great importance globally with countless millions of humans being infected or at risk of infection. Mathematical models have been used to calculate the risk of cancer due to infection; the calculated risk factor indicates that about 15% of all cases globally can be attributed to infections including those due to schistosomes and liver flukes. The infectious origin of a cancer implies that it is preventable. Therefore, if infections were prevented by increased educational efforts and improved public health initiatives, there would be considerably fewer cases of cancer in both developed and underdeveloped countries.”

The Lancet reports:

“Among parasitic diseases, infections with the two fish-borne liver flukes of the family Opisthorchiidae (trematodes), specifically Opisthorchis viverrini and Clonorchis sinensis, can induce cholangiocarcinoma, and infection with the blood fluke Schistosoma haematobium may cause cancer of the urinary bladder (Bouvard et al., 2009).”

 

 

Knowing this, it’s not a stretch to come to the conclusion that ridding the body of parasites could effectively prevent or treat some cancer. Many of us have heard about the Joe Tippens Protocol, where a man allegedly cured his late-stage cancer with an inexpensive veterinary product, a de-wormer called Fenbendazole. Experts like Dr. Bryan Ardis have weighed in on the discussion and concluded that anti parasitic drugs are an effective treatment with a documented history of success. He explains that approximately 50% of the cancerous tumors/polyps he saw with his patients had been misdiagnosed and were actually parasitic egg sacs. He indicated that 70% of autoimmune cases seen in his practice - were caused by parasites.

 

In a 2021 Johns Hopkins study published in the journal Oncotarget on July 6, Gregory Riggins, M.D., Ph.D., professor of neurosurgery and oncology at the Johns Hopkins University School of Medicine, and his team ‘used two different mouse models to determine that the anti-parasitic drug mebendazole could slow or stop the growth and spread of both early and late-stage pancreatic cancer. Riggins and his team administered mebendazole to mice that were genetically engineered to develop pancreatic cancer’. Read that again: they genetically manipulated them to develop cancer.

Riggins concluded: “We think that mebendazole could have a role in all stages. It was particularly effective for pancreatic cancer that was detected early.”

Oncologist Dr. William Makis has compiled data that shows that ‘Fenbendazole has at least 12 proven anti-cancer mechanisms in vitro and in vivo’. I encourage you to read his article where he goes in depth on each of the studies that support the following conclusions of anti-cancer mechanisms.

 

There is evidence to suggest that anti-parasitic medications treat the following cancers:

 

I will tell you simply that I do not believe that anything here conclusively proves that all cancer is parasitic but the questions are fair and legitimate. I also do not believe that FenBen, Ivermectin or anything else will be the magic pill that cures everything we call cancer. That said, the parasite hypothesis deserves legitimate and unbiased research and while I may not believe those two drugs will cure all cancer I absolutely believe they could have a therapeutic role and potentially cure some cancers. Ultimately I think it is fair to ask whether anti-parasitic drugs are showing promise in fighting some cancer because the cancer is a result of parasites or if it is because the cancer is susceptible to treatments for parasites.

Ultimately anti parasitic drugs should investigated to determine if and how they should be included in the treatment of cancer. Fenbendazole is just ONE of the drugs in this class with others like Ivermectin proving to be equally as promising. The real question is, will Big Pharma or our crooked government ever legitimately investigate this?

Sources:

1. Parasites that Can Lead to Cancer | American Cancer Society | American Cancer Society

2. brmedj04652-0016.pdf (nih.gov)

3. How Little Do Users Read? (nngroup.com)

4. Abstract 2313: Fenbendazole induces cell cycle arrest in colorectal cancer cells and patient-derived colon cancer organoids | Cancer Research | American Association for Cancer Research (aacrjournals.org)

5. The Overlooked Miracle Drug for Cancer? Why Big Pharma Fears Fenbendazole – Vigilant News Network

6. Joe Tippins' Fenbendazole Protocol For Cancer (deeprootsathome.com)

7. 

8. 

9. https://yournews.com/2023/08/24/2628420/dr-lee-merritt-cancer-tumors-are-parasites/

10. The Medical Rebel (drleemerritt.com)

11. Parasites--a New Paradigm (rumble.com)

12. Parasites: Types, Symptoms, Treatment & Prevention (clevelandclinic.org)

13. How Parasite Infection Can Lead to Cancer (naturalmentor.com)

14. Parasite-Derived Cancer | New England Journal of Medicine (nejm.org)

15. Cancer: An Unbiased Overview In Preparation for Our Upcoming War On Cancer (substack.com)

16. Parasite-microbe-host interactions and cancer risk | PLOS Pathogens

17. Antiparasitic Drugs: Malaria, Toxoplasmosis, Parasitic Infection (clevelandclinic.org)

18. How parasites get their HOOKs into human cells | Whitehead Institute (mit.edu)

19. Helminths in human carcinogenesis - ScienceDirect

20. Project MUSE - The Worm and the Tumor: Reflections on Fibiger's Nobel Prize (jhu.edu)

    Leave a comment

21. The Dr. Ardis Show (thedrardisshow.com)